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### Messages - sandokhan

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1
##### Flat Earth Theory / Re: Does observation of planets match Newton's and Kepler's laws?
« on: August 17, 2023, 07:29:56 PM »
I know RET better than you.

Please explain the faint young sun paradox. If you cannot, you are out of luck, your software simply uses the conventional RE diameters/distances for the planets in a PLANAR context. The orbits of the planets are not planar, they are helices on a cylinder.

Here is the faint young sun paradox:

https://www.theflatearthsociety.org/forum/index.php?topic=30499.msg1707290#msg1707290

2
##### Flat Earth Theory / Re: FE radius (UAFE estimate)
« on: August 17, 2023, 07:25:01 PM »
The answer to the question is related to the circumference of the Earth (Flat or Globe): it must give the same value in both versions.

https://archive.org/details/HapgoodCharlesHutchinsMapsOfTheAncientSeaKings/page/n27/mode/1up?view=theater (page 33 - the relationship between the value attributed to Erathosthenes and the radius of Piri Reis' map)

3
##### Flat Earth Theory / Re: FE radius (UAFE estimate)
« on: August 17, 2023, 05:39:36 PM »
Why does the shorter journey (Sydney - Jo'burg) take longer than a journey which appears to cover a much greater distance (Auckland - Santiago)?

Very simple: one journey is a straight line, while the other takes the airplane around Antarctica.

Now, we know for sure, absolutely 100%, that the radius of the Piri Reis map is 6,363.63 km, Charles Hapgood proved that fact while he had consulted with several noted mathematicians to help him out decipher the map.

Here is the question: we know that the Piri Reis map is an azimuthal equidistant projection (I believe the center is near the Marmara Sea, since the northern portion of the map is missing, but that is another matter). What, then, is the radius if the map would be projected onto a sphere?

4
##### Flat Earth Theory / Re: FE radius (UAFE estimate)
« on: August 17, 2023, 04:33:13 PM »

Look at the flight paths on a "globe" for Auckland-Santiago and Sydney-Johannesburg: on a FE map one of this routes would go between Antarctica and the Dome:

One flight path chooses to fly "above" Antarctica, while the other passes right between Antarctica and the Dome. Different durations of course.

Why did they choose the value of 6,363.63 km as the radius of the RE? Does it have anything to do some projection formula which would have worked if and only if 6,363.63 had been used?

Tesla had found out in 1908 that the map of Siberia was off by hundreds of kilometers. Most likely similar mistakes were involved in the distances attributed to Brazil, Argentina, Congo, Algeria, Lybia, Iran, Siberia, not to mention northern Canada.

5
##### Flat Earth Theory / Re: FE radius (UAFE estimate)
« on: August 17, 2023, 01:43:14 PM »
The 12 hr flight would take place in the most direct way seen on the Piri Reis map. Is there a problem? Two possible flight paths: above Antarctica (as seen on the map) or passing between Antarctica and the Dome.

Here are two of the most unusual maps you'll ever see:

https://stevedutch.net/pseudosc/1421.htm
https://stevedutch.net/pseudosc/piriries.htm

6
##### Flat Earth Theory / Re: FE radius (UAFE estimate)
« on: August 17, 2023, 11:26:42 AM »
The value of the radius is one of the most important elements of FET. If we know the radius, we can calculate the year as well as the month of the next reversal of the magnetic pole (the shift of the stellar dome as well).

Everyone on youtube, many other forums, is totally preoccupied with this issue, since it cannot be ignored anymore: when will the reversal of the magnetic poles take place? None of their dates can be justified, it is only FET which can offer a precise estimate.

You think that Auckland to Santiago de Chile was the most difficult route I had to deal with over the years? No, it was Juneau to Santiago de Chile:

https://www.theflatearthsociety.org/forum/index.php?topic=38712.msg961302#msg961302

7
##### Flat Earth Theory / Re: FE radius (UAFE estimate)
« on: August 17, 2023, 06:11:17 AM »
There is no such thing as Erathosthenes original data, since he had used spherical triangles. The FE radius must be 6,363.63 km, upper bound less than 10,000 km. This is what we are talking about.

8
##### Flat Earth Theory / Re: Does observation of planets match Newton's and Kepler's laws?
« on: August 17, 2023, 06:09:33 AM »
There is no such thing as Kepler's laws: Kepler had faked each and every entry, each calculation in Nova Astronomia:

https://forum.tfes.org/index.php?topic=10175.msg160200#msg160200

The 3D galactic orbit of the solar system looks like this:

https://i.postimg.cc/nL6hx23b/safari.jpg

What you'd need is a three dimensional Kepler law, something no one has ever derived or even thought of.

The 3D helical orbits of the planets cannot be justified, since one would need a lateral kind of gravitation which would pull the celestial bodies in such a manner.

9
##### Flat Earth Theory / Re: FE radius (UAFE estimate)
« on: August 17, 2023, 05:23:51 AM »
Whoever wrote the works attributed to Erathosthenes used spherical triangles:

https://web.archive.org/web/20080214044528im_/http://geocities.com/levelwater/eratostenesegments.gif
https://web.archive.org/web/20080214044528im_/http://geocities.com/levelwater/tangentedecurva02.gif
https://web.archive.org/web/20080214044528im_/http://geocities.com/levelwater/siennaalejandria.gif

That is not even the main point.

The FE map relates to the RE globe mathematically as a projection.

There is no globe since you cannot justify how four trillion billion liters of water stay in place next to the outer surface of a sphere.

Take a look at this, half of the surface area of the globe is occupied by the Pacific Ocean:

By contrast, the true flat earth Piri Reis map looks like this:

https://www.vhv.rs/dpng/d/449-4490274_theflatearthsociety-org-forum-sandokhan-piri-reis-map-hd.png

The surface area of the globe is four times as large as the surface area of the FE map.

How was it done?

https://web.archive.org/web/20210116142615/https://civilengineering-softstudies.com/2018/04/error-and-correction-for-the-curvature-of-the-earth-and-refraction-surveying.html

http://web.pdx.edu/~i1kc/courses/Surveying/Handouts/JGE%20Paper%201%20-%20Introduction.pdf (pg 14, curvature and refraction)

For the past 220 years, no matter which country, the CORRECTION for the supposed curvature of the Earth was added, automatically, for each and every surveying/topographical mission.

But here is something no one else has ever thought of: since there will always be some misalignment between the source and the reflector, the strings of light used by the theodolite will be subject to the influence of the Coriolis effect. That is, the path of the light strings will be deflected. This fact is not taken into consideration by land surveyors at all.

The surveying was done on land of course, and since they needed a huge surface area for the Pacific Ocean, they simply added it on maps with no problem.

Why is the radius of the flat earth so important? Because it tells us immediately when the next magnetic pole shift will take place. Since s = r x θ and we can't do anything about the angle [θ = 0.959582 rad (2 x 27.49 = 54.98, 54.98° = 0.959582 rad, 1/27.49 = 0.036376864 = 0.1 - 0.063623), Tropic to Tropic (solstice to solstice)], only the value of the radius is in question. I believe the radius of the FE measures 6,363.63 km.

The westward precessional shift of the Sun (1.5 km/year or 4.2 meters/day) must be taken into account.

The distances on the supposed globe have been increased greatly, see the distortions in Antarctica as an example:

http://www.moonglow.net/eclipse/2003nov23/

The photographers must be located some few hundred kilometers from the Sun (height of 636 meters), not the thousand of kilometers which would be assumed to be correct; given this fact, the Apollo 11 mission astronauts might as well have taken a sledge with reindeers to the Moon.

10
##### Flat Earth Theory / FE radius (UAFE estimate)
« on: August 11, 2023, 07:04:34 PM »
https://wiki.tfes.org/Eratosthenes_on_Diameter

Therefore we can take the distance of 500 miles, multiply by 25, and find that the radius of the flat earth is about 12,250 miles. Doubling that figure for the diameter we get a figure of 25,000 miles.

12,250 mi = 19,600 km

This can't be right, not even close.

The lower bound would be R = 6,363.63 km, the upper bound (the RE distance from the equator to the NP) is 10,000 km.

More details here:

http://www.ilya.it/chrono/pages/erdmessungen.htm

11
##### Philosophy, Religion & Society / Re: Died Suddenly
« on: January 06, 2023, 03:05:26 PM »
Covid-19 is an airborne M. avium. Had each person, in each country, received a kit with clarithromycin/azithromycin through mail, back in february of 2020, there would have been no pandemic at all.

Now, the world is totally unprepared to deal with Coronapox and Coronaflu. Omicron has already activated its prion domain.

https://www.panspermia.org/whatsnew91.htm#20180207

More than 800 million viruses per square meter per day descend from the high atmosphere to the ground.
bacteria and viruses in Spain. This news comes from an international team who sampled the air weekly or bi-weekly from two mountain sites 3 km high in southern Spain. They found that bacteria and viruses above the atmospheric boundary layer are more abundant, can persist longer and can travel farther than previously known.

No one is asking the most important question: why has the volume of such pathogens increased by a large factor ever since November of 2019? Certainly these mycobacteria arrive each and every day on the surface of the Earth.

The lethal wave of influenza in 1918-19... was first detected on the same day in Boston and Bombay. Yet in spreading within the United States it took three weeks to go from Boston to New York. — Fred Hoyle and Chandra Wickramasinghe

https://www.panspermia.org/panfluenza.htm

https://vixra.org/pdf/2002.0118v1.pdf

https://www.panspermia.org/whatsnew99.htm#20200227

https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(03)13440-X/fulltext

https://www.panspermia.org/virusesfromspace2.pdf

On October 11 2019 a meteoritic bolide (probably fragment of a comet) explodes in a brief flash in Nth East China. We think it probable that this bolide contained embedded within it a monoculture of infective nCoV-2019 virus particles that survived in the interior of the incandescent meteor.

Also well documented is that, in the winter of 1918, the disease appeared suddenly in the frozen wastes of Alaska, in villages that had been isolated for several months. Mathematical modelling of epidemics such as the one described invariably involves the ad hoc introduction of many unproven hypotheses—for example, that of the superspreader. In situations where proven infectivity is limited only to close contacts, a super-spreader is someone who can, on occasion, simultaneously infect a large number of susceptible individuals, thus causing the sporadic emergence of new clusters of disease. The recognition of a possible vertical input of external origin is conspicuously missing in such explanations.

With respect to the SARS outbreak, a prima facie case for a possible space incidence can already be made. First, the virus is unexpectedly novel, and appeared without warning in mainland China. A small amount of the culprit virus introduced into the stratosphere could make a first tentative fall out East of the great mountain range of the Himalayas, where the stratosphere is thinnest, followed by sporadic deposits in neighbouring areas. If the virus is only minimally infective, as it seems to be, the subsequent course of its global progress will depend on stratospheric transport and mixing, leading to a fall out continuing seasonally over a few years. Although all reasonable attempts to contain the infective spread of SARS should be continued, we should remain vigilant for the appearance of new foci (unconnected with infective contacts or with China) almost anywhere on the plant. New cases might continue to appear until the stratospheric supply of the causative agent becomes exhausted.

https://cosmictusk.com/wickramasinghe-predicted-coronavirus-pandemic-in-november-2019/

On November 25th, 2019, Professor Chandra Wickramasinghe made the following stark warning, weeks before the coronavirus emerged.

On the basis of this data, there appears to be a prima facie case for expecting new viral strains to emerge over the coming months and so it would be prudent for Public Health Authorities the world over to be vigilant and prepared for any necessary action. We need hardly to be reminded that the spectre of the 1918 devastating influenza pandemic stares us in the face from across a century.

Chandra Wickramasinghe, Current Science, November 25, 2019

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1079424/

https://www.researchgate.net/publication/326160954_Comets_and_Contagion_Evolution_Plague_and_Diseases_From_Space

http://journalofcosmology.com/Panspermia10.html

https://www.longdom.org/open-access/the-role-of-viruses-and-viral-infections-in-the-theory-of-panspermia-2332-2519.1000111.pdf

Would it still be possible to achieve herd immunity, even at this late stage of the pandemic? Yes. If the health authorities in each country would distribute clarithromycin to each person, herd immunity could still be achieved.

12
##### Philosophy, Religion & Society / Re: Died Suddenly
« on: January 06, 2023, 09:03:17 AM »
And some iron plus infralasers shouldn't do anything.

But they will.

You are forgetting what happened during the month of March, 2021, in eastern Europe (as an example). All of a sudden, the new exotic variants B.1.1.7, P.1, B.1.351 had emerged all over the place, without any travel history on the part of the infected persons. Using the spike proteins and HeLa cells (for AZ/J vaccines), the mycobacterium did send electromagnetic copies of their cells to the bacteria in the atmosphere, which promptly received the new signal and changed their configuration to reflect the new variants which had been developing in the vaccinees.

Vaccinations do cause strains.

https://www.francesoir.fr/opinions-tribunes/covid-19-questions-sur-les-vaccins

Mais patatras, voilà que l’Académie de médecine s’y met, et maladroitement, car son explication pour ne pas différer la seconde injection, remet dans le circuit le risque de mutations dues aux vaccins. Outre le fait que « le retard peut faire en sorte que des anticorps facilitants créés pourrait exacerber la Covid-19 », le communiqué indique aussi que « le faible niveau d’immunité (après la primo-vaccination) constituera un terrain favorable pour sélectionner l’émergence d’un ou de plusieurs variants échappant à l’immunité induite par la vaccination ». Cela a été répété par Yves Buisson rapporteur, sur France Info.

Seconde question : y a-t-il un lien entre vaccins et derniers mutants ? Le communiqué de l’Académie de médecine dit que c’est possible, et des faits me troublent.

By spacing the two doses of vaccine, "we risk having mutations" of the virus, alerted Tuesday, January 12 on franceinfo, Professor Yves Buisson of the National Academy of Medicine, president of the Covid-19 cell .

In addition to the fact that "the delay may ensure that facilitating antibodies created could exacerbate Covid-19", the press release also indicates that "the low level of immunity (after the primary vaccination) will constitute a favorable ground for selecting the 'emergence of one or more variants escaping immunity induced by vaccination'.

Second question: is there a link between vaccines and the latest mutants? The press release from the Academy of Medicine says it is possible, and I am troubled by the facts.

13
##### Philosophy, Religion & Society / Re: Died Suddenly
« on: January 06, 2023, 07:00:30 AM »
Plus, the sigal strength is going to be very very small.  So how would it reach into the air to "Talk" to anything?  And how does it control the random electron movement?

Exactly. So, how would you make sure that those bacteria benefit from using both an amplifier and an antenna? Remember this?

https://web.archive.org/web/20210611184613/ https://www.youtube. com/watch?v=lXjjOLhFnHA (remove the spaces)

Twitter from "Trump" in 2017: "covfefe".

Covid ferritin Fe cages. A mobile phone is a non-metallic object. So, you'd need an infrared laser and the quantum Hall effect.

14
##### Philosophy, Religion & Society / Re: Died Suddenly
« on: January 05, 2023, 06:24:03 PM »
K. Blome had discovered that bacteria communicate wirelessly. In 2009, Nobel prize winner Luc Montagnier confirmed these experiments. Then, in 2011, finally the finishing touches and theoretical results were added:

https://arxiv.org/pdf/1104.3113.pdf

Electromagnetic Signals from Bacterial DNA

But the most interesting work done on Blome's original experiments was performed by Dr. Vlail Kaznacheyev:

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4267444/

https://www.researchgate.net/publication/318985550_Heuristic_View_on_Quantum_Bio-Photon_Cellular_Communication

https://pdfcoffee.com/the-kaznacheyev-experiments-2-pdf-free.html

15
##### Philosophy, Religion & Society / Re: Died Suddenly
« on: January 05, 2023, 05:18:35 PM »

https://arxiv.org/pdf/1104.3113.pdf

Montagnier's experiment can be summarised as follows:

A known water sample with 2 ng/ml of 104 bases DNA from an HIV infected patient is diluted by 10 into water and agitated for 15 seconds. After filtration to remove the DNA, the dilution and agitation steps are repeated 10 times, reaching high dilution levels of 10^−10.

The highly diluted sample emits electromagnetic signals (EMS) of low frequencies.

This EMS is recorded by a microphone coil and saved as a 6-second WAV file at the lab in Paris.

The WAV file is emailed to a partner team at the university of Benevento in Italy.

The Italian team emits with a coil for 1 hour the EMS of the WAV file on a sample of distilled water in a sealed metal tube.

The water sample is then placed in a polymerase chain reaction (PCR) machine.

The PCR machine in Italy produces DNA, 98% identical to the initial DNA in Paris.

The experiment was first made in July 2005, and was repeated and filmed for a TV documentary in 2013, released on the French channel France 5 on 5 July 2014.

https://arxiv.org/abs/1501.01620

Transduction of DNA information through water and electromagnetic waves

https://arxiv.org/pdf/1501.01620.pdf

The quantum field theoretical analysis of the phenomenon points to the crucial role played by coherent molecular dynamics.

The problem has been that Montagnier showed that when compared to pure water, samples chockfull of bacteria, emitted more radio waves, and no one could explain why.

Researchers have known for years that some bacteria do communicate via nanowires, which led Widom and his team to conclude that it wasn’t so farfetched to believe more highly developed bacteria, such as E. coli or Mycoplasma pirum, might instead communicate via wireless medium.

It’s likely these new findings will incite others to look a little deeper, however, as the main argument for rejecting Montagnier’s findings back in 2009, was that bacteria lacked a means for generating radio signals; an assertion that has now been overthrown.

More importantly, though, if simple organisms can communicate using radio waves — and have been communicating using radio waves for billions of years — it would shake the entire bedrock of modern science. Montagnier’s work suggests that cells can send electromagnetic imprints of itself to other, remote cells, but why stop there? If human cells also communicate using radio waves, we might be able to create a digital, silicon-based interface for ‘hacking’ our physiological infrastructure.

Montagnier’s work suggests that cells can send electromagnetic imprints of itself to other, remote cells.

The principle is similar to Benveniste's experiment from 1997[12] where EMS was recorded from ovalbumine at the Northwestern University Medical School of Chicago, and transmitted through email to Benveniste's Digital Biology Laboratory in Clamart, France.

After emitting the signal on pure water for 20 minutes, the water could cause an allergic shock on an isolated Guinea-pig heart allergic to ovalbumine. In both experiments the EMS reproduces the properties of the original molecules in their absence.

https://www.jacionline.org/article/S0091-6749(97)81064-0/pdf (pg 75 of the pdf document, item 705)

Omicron is attaining its original, virulent form, M. bovis; right now, it is at the M. influenzae stage, where it begins to activate its prion domain. As an example, C.H.11 has the P681R mutation of Delta.

That is why those experiments in the lab (Omicron + Wuhan) are so dangerous. Those mycobacterium will start to transmit copies of their cells to the M. avium/M. influenzae in the atmosphere, that is how all of the variants had appeared everywhere so fast, almost at once, simultaneously.

16
##### Philosophy, Religion & Society / Re: Died Suddenly
« on: January 05, 2023, 01:52:34 PM »
Spike proteins had been discovered some one hundred years ago by Wilhelm Reich. He called them T-bacilli. Here is a diagram, which had been drawn by Reich himself, of the T-bacilli:

If T-bacilli appeared as “spikes” projecting out of the disintegrating cells during their disintegration, Reich considered this a still more serious indicator of incipient biopathic disease.

The medical term for red blood cells with T-bacilli is Poikilocytosis (term for abnormal shaped red blood cells in the blood; Acanthocytes - red blood cells with unevenly spaced spiked projections that have blunted ends).

Spike proteins are liquid crystals.

T-bacilli are dextrorotatory prions (PrPSc), or beta sheet prions. Beneficial prions are alpha helix or PrPC prions (laevorotatory prions/peptides).

Antidote: 1. alpha helix prions/peptides, 2. proteolytic enzymes (such as bromelaine), 3. specific antibiotics for the mycobacterium.

At the same time that W. Reich was investigating spike proteins, Kurt Blome had discovered the existence of HeLa cells, and that bacteria communicate through radio waves. Some fifteen years later, this technology was imported to the US. Both spike proteins and HeLa cells can send electromagnetic copies of the bacteria from the laboratory to the mycobacterium in the atmosphere.

How did D614G replace Wuhan in just two weeks, worldwide? How did the exotic variants, B.1.1.7, P.1, B.1.351 appear all of a sudden in eastern Europe, with no travel history? Exactly, the primary mode of transmission of Covid-19 is: atmosphere-person. That is why masks are useless. M. avium is handled in BSL-3 level labs, imagine to enter those facilities wearing a cloth mask. It is not even a laughing matter. The secondary mode of transmission is person to person (a minute number of the total cases).

The 1918 flu had started back in 1910 with the arrival of the pathogens from comet Halley (through the Beta Taurids and Taurids meteor showers), that is when a huge pandemic had erupted in China (Himalaya constitutes the first major barrier for those pathogens which are raining down from the atmosphere). Then Chinese workers brought this disease to Europe. During 1915-1917, there was  worldwide pandemic of "coronavirus" (actually M. avium). In 1918, M. avium became M. influenzae. The catalyst was the eruption of the Katla volcano, in October 12, 1918. In turn, M. influenzae became M. bovis (T-bacilli prions).

That is how I knew back in September of 2021 that Omicron (M. influenzae) was going to replace M. avium (Sars-Cov-2). Omicron uses the DPP4 cellular receptor, same as Mers-Cov. The R0 factor for Mers-Cov was the highest ever recorded (higher than measles): 7-19 (in South Korea as an example). Omicron is Mers-Cov-2, and is activating its prion (T-bacilli) domain/region. What Omicron needed, in order to activate these prions, was a helper, another pathogen, MPV. MPV is mousepox, and poxviridae is a variant of M. leprae. There is common genome between Omicron and MPV (amino acid chains of Plasmodium falciparum and Yersinia pestis). MPV is caused by non-canonical RNA G-quadruplexes configurations. MPV contains amino acid sequences from the simian hemorrhagic fever pathogen.

Coronathrax - the pathogen of comet Encke, which will arrive after the reversal of the magnetic poles.

Coronapox - the reassortment of MPV and Omicron (their respective RNA segments).

Coronaflu - the activation of the prion domain in Omicron.

17
##### Philosophy, Religion & Society / Re: Died Suddenly
« on: December 27, 2022, 02:11:21 PM »
Dr. Broxmeyer could not have published medical data in the Oxford Journal of Infectious Diseases unless his credentials had been checked and verified. This alone disproves your vitriolic attack against a scientist whose articles on infectious diseases speak for themselves. All of his other articles have been published in medical journals, and thus have been peer-reviewed. Do not confuse the stated reasons for the revocation of the medical license with the hidden agenda which was aiming at his unwaivering support for the bacterial cause of various diseases.

Obviously you do not know about his work on bacterial infections. He is saying that HIV is also a mycobacterium, certainly not a virus.

https://www.sidastudi.org/resources/inmagic-img/dd7314.pdf

Your doctor obviously did not test for M. avium, as these tests can be performed only by real experts on bacterial infections, such as Dr. Broxmeyer. Cell wall deficient mycobacterium look just like a virus and they pass through the same filters.

You have some explaining to do here. Certainly the bacterial epitopes (including M. avium) which have found on the spike protein of Sars-Cov-2 confirm the work published by L. Broxmeyer.

Out of nine sites, seven sites showed molecular similarity with 54 antigenic determinants found in twelve pathogenic bacterial species (Mycobacterium tuberculosis, Mycobacterium leprae, Bacillus anthracis, Borrelia burgdorferi, Clostridium perfringens, Clostridium tetani, Helicobacter Pylori, Listeria monocytogenes, Staphylococcus aureus, Streptococcus pyogenes, Vibrio cholera and Yersinia pestis), two malarial parasites (Plasmodium falciparum and Plasmodium knowlesi) and influenza virus A.

Most of the bacterial antigens that displayed molecular similarity with antigenic sites in SARS-CoV-2 RBD (receptor binding domain) were toxins and virulent factors. Antigens from Mycobacterium that showed similarity were mainly involved in modulating host cell immune response and ensuring persistence and survival of pathogen in host cells.

https://www.sciencedirect.com/science/article/pii/S0171298521000395

Sars-Cov-2 also features epitopes from M. bovis, Nipah, and other pathogens.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7423587/

BlastP analysis showed high homology of the SARS-CoV-2 envelope protein with 12 consecutive amino acids of the protein LytR C, which is a consensus protein unique to Mycobacteria.

https://www.ncbi.nlm.nih.gov/labs/pmc/articles/PMC8577642/

However, the knowledge that heat shock protein (HSP)65 is the main antigen of Mycobacterium bovis BCG prompted us to verify whether sequence similarity existed between HSP65 and SARS-CoV-2 spike (S) and nuclear (N) proteins that could support an antigen-driven immune protection of BCG vaccine. The results of the in silico investigation showed an extensive sequence similarity of HSP65 with both the viral proteins, especially SARS-CoV-2 S, that also involved the regions comprising immunodominant epitopes.

Had Dr. Broxmeyer's BCG vaccines with bacteriophages specific for M. avium been implemented, Covid-19 would have been over in the month of february of 2020. Had Sars-Cov-2 been identified correctly as M. avium, the correct treatment (clarithromycin or even solithromycin) could have been administered from the start. Yet, cmRNA isomeric vaccines were put forth as treatment, but these vaccines are coded with Pseudouridine, a chiral isomer, which will only force the immune system to produce isomeric abs which have nothing to do with Sars-Cov-2.

18
##### Philosophy, Religion & Society / Re: Died Suddenly
« on: December 27, 2022, 08:38:48 AM »
Here is the cmRNA genetic code:

https://web.archive.org/web/20210111092707/https://berthub.eu/articles/posts/reverse-engineering-source-code-of-the-biontech-pfizer-vaccine/

Uracil has been replaced 100% with Pseudouridine (Ψ), which is a chiral isomer. This means that the cmRNA genetic code is for an ISOMERIC Sars-Cov-2, and has nothing to do with Sars-Cov-2 which is coded with Uracil. As such, the immune system will produce ISOMERIC antibodies (isomeric abs were discovered in 1994), which have nothing to do with the pathogenic agent Sars-Cov-2.

https://anandamide.substack.com/p/differences-in-vaccine-and-sars-cov

https://www.mdpi.com/2076-393X/9/7/734/htm

However, superficial application of these two criteria can lead to mistakes. I will take the CGN codon family for Arg to show an incorrect optimization of the two mRNA vac.cines.

The designers of both vac.cines considered CGG as the optimal codon in the CGN codon family and recoded almost all CGN codons to CGG. There are two lines of evidence suggesting that CGG is not the optimal codon. These multiple lines of evidence suggest that CGC is a better codon than CGG. The designers of the mRNA vac.cines (especially mRNA-1273, Table 1) chose a wrong codon as the optimal codon.

Pfizer/BioNTech’s BNT162b2 mRNA features two consecutive UGA stop codons. Moderna’s mRNA-1273 uses all three different stop codons UGAUAAUAG. Are these the optimal arrangement?

With such a +1 frameshifting, a downstream in-frame stop codon cannot serve as a fail-safe mechanism. UGA is a poor choice of a stop codon, and UGAU in Pfizer/BioNTech and Moderna mRNA vac.cines could be even worse.
One caveat in the reasoning above involves the replacement of U by N1-methylpseudouridine (Ψ) in the two vac.cine mRNAs.

Therefore, the stop signals are ΨGAΨGA instead of UGAUGA in Pfizer/BioNTech’s vac.cine, and ΨGAΨAAΨAG instead of UGAUAAUAG in Moderna’s vaccine. As Ψ is more promiscuous in base-pairing than U and can pair with both A and G and, to a less extent, with C and U, stop codons become more prone to misreading by tRNAs. It is for this reason that both mRNA vaccines use consecutive stop codons as a fail-safe mechanism, with the hope that no frameshifting occurs when the first stop codon fails. However, UGAU is known to cause a +1 frameshifting. It is reasonable to infer that ΨGAΨ may be the same. I have mentioned before that mammalian AZ1 gene with a stop codon context UGAU is prone to polyamine-induced +1 frameshifting. Such a +1 frameshifting defeats the purpose of having multiple stop codons as a fail-safe mechanism.

19
##### Philosophy, Religion & Society / Re: Died Suddenly
« on: December 27, 2022, 06:52:22 AM »
No, he got suspended because he would not waiver on the matter that all of the ailments that you had described were not viruses. I had done my homework, since everything you posted was well-known even two years ago, in the other thread which ran into 39 pages of debate.

All of Dr. Broxmeyer's papers are peer-reviewed. Especially this one, which was published in the most prestigious medical journal in the world:

Broxmeyer, L., Sosnowska, D., Miltner, E., Chacon, O., Wagner, D., McGarvey, J., Barletta, R.G. and Bermuddez, L.E. (2002) Killing of Mycobacterium avium and Mycobacterium tuberculosis by a mycobacteriophage delivered by a nonvirulent mycobacterium: a model for phage therapy of intracellular bacterial pathogens. J Infect Dis 186,
1155–1160

The Journal of Infectious Diseases

Killing of Mycobacterium avium and Mycobacterium tuberculosis by a mycobacteriophage delivered by a nonvirulent mycobacterium: a model for phage therapy of intracellular bacterial pathogens.

We are currently exploring the use of other mycobacteriophages and attenuated mycobacterial strains of M. avium and M. tuberculosis, as well as bacille Calmette-Guerin as potential phage delivery systems.

All of his papers which describe zika, ebola, H1N1, as having been caused by mycobacteria are peer-reviewed. That is why he is a very credible source.

What about the credibility of the medical doctors you believe in?

This test cannot rule out diseases caused by other bacterial or viral pathogens.

Positive results are indicative of active infection with 2019-nCoV but do not rule out bacterial infection or co-infection with other viruses. The agent detected may not be the definite cause of disease.

Always, one needs to test for BOTH viruses and mycobacteria. Did your medical doctors perform these tests? All of them have refused to test for mycobacteria.

But not L. Broxmeyer.

Can you explain to your readers why Sars-Cov-2 has so many bacterial epitopes?

Out of nine sites, seven sites showed molecular similarity with 54 antigenic determinants found in twelve pathogenic bacterial species (Mycobacterium tuberculosis, Mycobacterium leprae, Bacillus anthracis, Borrelia burgdorferi, Clostridium perfringens, Clostridium tetani, Helicobacter Pylori, Listeria monocytogenes, Staphylococcus aureus, Streptococcus pyogenes, Vibrio cholera and Yersinia pestis), two malarial parasites (Plasmodium falciparum and Plasmodium knowlesi) and influenza virus A.

Most of the bacterial antigens that displayed molecular similarity with antigenic sites in SARS-CoV-2 RBD (receptor binding domain) were toxins and virulent factors. Antigens from Mycobacterium that showed similarity were mainly involved in modulating host cell immune response and ensuring persistence and survival of pathogen in host cells.

https://www.sciencedirect.com/science/article/pii/S0171298521000395

20
##### Philosophy, Religion & Society / Re: Died Suddenly
« on: December 26, 2022, 05:10:50 PM »
You haven't done your homework on the subject, which is a pity.

Influenza A, ebola, zika, rhinoviruses are mycobacteria. Azithromycin is used especially for M. avium.

https://web.archive.org/web/20180508034750/http://drbroxmeyer.netfirms.com/001pdfBIRDFLUEDITORIALPUBLISHED.pdf

http://www.oilgeopolitics.net/Swine_Flu/Tuberculosis/tuberculosis.html

Out of nine sites, seven sites showed molecular similarity with 54 antigenic determinants found in twelve pathogenic bacterial species (Mycobacterium tuberculosis, Mycobacterium leprae, Bacillus anthracis, Borrelia burgdorferi, Clostridium perfringens, Clostridium tetani, Helicobacter Pylori, Listeria monocytogenes, Staphylococcus aureus, Streptococcus pyogenes, Vibrio cholera and Yersinia pestis), two malarial parasites (Plasmodium falciparum and Plasmodium knowlesi) and influenza virus A.

Most of the bacterial antigens that displayed molecular similarity with antigenic sites in SARS-CoV-2 RBD (receptor binding domain) were toxins and virulent factors. Antigens from Mycobacterium that showed similarity were mainly involved in modulating host cell immune response and ensuring persistence and survival of pathogen in host cells.

https://www.sciencedirect.com/science/article/pii/S0171298521000395

Sars-Cov-2 is a mycobacterium, and so is Omicron (Mers-Cov-2).

The first major result on the use of cmRNA vaccines:

https://unglossed.substack.com/p/boosting-tolerance-igg4

I would not call it tolerance. IgG4 leads to desensitization. Ongoing exposure to spike can lead to IgG4 related disease (IgG4-RD). IgG4 induction is the second stage. It is most likely preceded by IgE induction. Injecting any antigen will induce IgE. I warned against it. I also wanted them to measure IgG1,2,3,4. Finally someone has done it.

https://www.researchgate.net/publication/279299679_An_Atlas_of_RNA_Base_Pairs_Involving_Modified_Nucleobases_with_Optimal_Geometries_and_Accurate_Energies

It is interesting that, when focusing on the H-bonded bases, the Pseudouridine modification seems rather to have a destabilizing than a stabilizing effect.

https://www.ncbi.nlm.nih.gov/labs/pmc/articles/PMC6009661/

Misincorporation of pseudouridine by T7 RNA polymerase can have implications for RNA-based therapeutics, as pseudouridine is incorporated into RNA to reduce immunogenicity.

https://elifesciences.org/articles/60917

A prion accelerates proliferation at the expense of lifespan

https://www.ncbi.nlm.nih.gov/labs/pmc/articles/PMC2965242/

Effect of pseudouridylation on the structure and activity of the catalytically essential P6.1 hairpin in human telomerase RNA

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